The results demonstrated that Mndoped ZnO/Graphene nanocomposites

The results demonstrated that Mndoped ZnO/Graphene nanocomposites efficiently bleached out the MB, showing an impressive photocatalytic enhancement over Mn-doped ZnO and pure ZnO samples. The enhanced activity of composite photocatalysts can be attributed to large adsorption by the dyes, enhanced visible light absorption and efficient charge separation

and fast transfer processes. (C) 2014 Elsevier Ltd and Techna Group S.r.l. All rights reserved.”
“ObjectiveThis study examined the mechanisms by which H2S modulates coronary microvascular resistance and myocardial perfusion at rest and in response to cardiac ischemia. MethodsExperiments were conducted in isolated coronary arteries and in open-chest anesthetized dogs. ResultsWe found that the H2S substrate l-cysteine (1-10mM) did not alter coronary selleck compound tone of isolated arteries in vitro or coronary blood flow in vivo. In contrast, intracoronary (ic) H2S (0.1-3mM) increased coronary flow from 0.490.08 to 2.65 +/- 0.13mL/min/g (p smaller than 0.001). This increase in flow was unaffected by inhibition of K-v channels with 4-aminopyridine (p=0.127) but was attenuated (0.23 +/- 0.02-1.13 +/- 0.13mL/min/g) by the K-ATP channel antagonist glibenclamide (p smaller than Repotrectinib 0.001). Inhibition of NO synthesis (l-NAME) did not attenuate coronary responses to H2S. Immunohistochemistry revealed expression of CSE, an endogenous H2S enzyme, in myocardium. Inhibition

of CSE with -cyano-l-alanine (10M) had no effect on baseline coronary flow or responses to a 15-second coronary occlusion (p=0.82). ConclusionsThese findings demonstrate that exogenous H2S induces potent, endothelial-independent dilation of the coronary microcirculation predominantly through the activation of K-ATP channels, however, our data do not support a functional role for endogenous H2S in the regulation of coronary microvascular resistance.”
“BACKGROUND: Individuals with multiple sclerosis (MS) experience some of the highest unemployment rates among all groups of chronic illnesses. Pain has been found to be a common reason

for sick leave or early retirement in healthy populations or other groups with find more chronic illness; however, there is little awareness regarding the effect of pain on the work status of individuals with MS. OBJECTIVES: To estimate the extent to which individuals with pain differ in employment status compared with those without pain among MS patients. METHODS: An extensive systematic review of the scientific literature was performed within the framework of the Cochrane Collaboration to identify studies focusing on the effect of pain on employment in individuals with MS. The following databases were searched: PubMed, EMBASE, PsychInfo, Web of Science, MD Consult and Elsevier, and Science Direct. The methodological quality of studies was assessed using the McMaster Critical Review Form. RESULTS: Ten articles met the inclusion criteria and were included in the systematic review.

Comments are closed.