The patient was followed up closely, and her abdominal pain subsided spontaneously. However, she was incidentally found to have isolated hepatic calcification, which may have been due to hypoparathyroidism. Discussion Hepatic
calcification is a rare event which usually occurs as a result of inflammatory conditions. The main causes of Inhibitors,research,lifescience,medical hepatic calcification are infections-e.g. tuberculosis, histoplasmosis, brucellosis, schistosomiasis, hydatid cyst, cytomegalovirus, toxoplasmosis, Pneumocystis carinii selleck inhibitor infection, chronic amebic or pyogenic abscess, and chronic granulomatous disease of childhood. Vascular problems-including hepatic artery aneurysm, portal vein thrombosis, and hematoma as well as neoplastic processes such as hemangioma, hepatocellular adenoma and carcinoma, Inhibitors,research,lifescience,medical infantile hemangioendothelioma, cholangiocarcinoma, hepatoblastoma, and metastatic tumors of the liver represent Inhibitors,research,lifescience,medical the remaining
etiologies.5 Diffuse hepatic calcification is seen even more rarely and the differential diagnosis is narrower. It usually occurs after ischemic insult in patients with end-stage renal disease on hemodialysis and as a sequella of shock liver.6,7 To find out the cause of diffuse hepatic Inhibitors,research,lifescience,medical calcification, we should rule out other differential diagnosis in each case. We evaluated liver and renal function tests, fasting blood glucose, calcium, phosphorus, prothrombin and partial thromboplastin times, thyroid function, tuberculin skin test, serology for brucella infection, hepatitis B and C, anti-cytomegalovirus IgM and IgG, human immunodeficiency virus, serology for toxoplasma infection,
and workup Inhibitors,research,lifescience,medical for hydatid cyst and amebic and fungal infections to rule out renal failure. Additionally, we evaluated infections such as tuberculosis, histoplasmosis, brucellosis, JNK-IN-8 schistosomiasis, hydatid cyst, cytomegalovirus, toxoplasmosis, Pneumocystis carinii infection, chronic amebic or pyogenic abscess, and chronic granulomatous disease of childhood. All the tests were normal and showed no hint of infection. Also, level of alfa feto protein, serum BHCG, abdominal sonography, portal and hepatic vein Doppler sonography, and abdominal spiral CT scan with intravenous and oral contrast were conducted and revealed no clue for vascular problems-including portal vein thrombosis and hematoma-as well as neoplastic processes such as hemangioma, hepatocellular adenoma and carcinoma, infantile hemangioendothelioma, cholangiocarcinoma, hepatoblastoma, and metastatic tumors of the liver.